NetaRosezweig
@NetaRose11
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organoids, while a powerful tool for studying human biology, lack immune systems, which is a significant limitation. NIH prioritized organoids to model human tissues and diseases in vitro. However, their lack of an immune system hinders their ability to fully recapitulate human
ICYMI: #NIH announces a new initiative to reduce animal use in research by launching ORIVA, a new office dedicated to advancing non-animal methods. Read more in the Washington Update: https://t.co/Dkl4zwjTok
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Thrilled to share invited review with the brilliant @NetaRose11! in the @ImmunityCP special issue on neuroimmune interactions. We explore shared immune pathways in Alzheimer’s & AMD, and chart new directions for cross-disease therapeutic strategies. https://t.co/rBSe7ALa52
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Online now: Alzheimer’s disease and age-related macular degeneration: Shared and distinct immune mechanisms
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Common immune system mediated threads of Alzheimer's disease and age-related macular degeneration, a new open-access review https://t.co/R57sSvvBQn
@ImmunityCP
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APOE4-dependent neutrophil-microglia interactions drive cognitive impairment in female individuals with Alzheimer’s disease via IL17F @ButovskyLab, @OlegButovsky, @NetaRose11, @KilianKleemann #neutrophils, #microglia, #IL-17 https://t.co/mH5PoQn3FG
nature.com
Nature Medicine - APOE4-dependent neutrophil–microglia interactions drive cognitive impairment in female carriers with Alzheimer’s disease via IL-17F.
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Research Spotlight: Uncovering How a Cellular Miscommunication Leads to Cognitive Impairment in Female Patients with Alzheimer’s Disease https://t.co/tHgegmwynl
@ButovskyLab @OlegButovsky @NetaRose11 @KilianKleemann
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Sex-Dependent APOE4 Neutrophil–Microglia Interactions Drive Cognitive Impairment in Alzheimer’s Disease https://t.co/NthVucSSp9 via @Science News
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This work led by Neta Rosenzweig (@NetaRose11), @KilianKleemann, & Thomas Rust provides a translational basis to target IL-17F in APOE ε4 female carriers with cognitive impairment. 🧠 Congratulations to all the authors! @OlegButovsky @EggenBart
https://t.co/ng9NXffRou
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@NatureMedicine @ButovskyLab @OlegButovsky @NetaRose11 @KilianKleemann Any Clinical development incoming of bimekizumab for Alzheimer Disease @UCB_France ?
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Sex-dependent APOE4 neutrophil–microglia interactions drive cognitive impairment in Alzheimer disease
nature.com
Nature Medicine - APOE4-dependent neutrophil–microglia interactions drive cognitive impairment in female carriers with Alzheimer’s disease via IL-17F.
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APOE4-dependent neutrophil-microglia interactions drive cognitive impairment in female individuals with Alzheimer’s disease via IL17F @ButovskyLab, @OlegButovsky, @NetaRose11, @KilianKleemann #neutrophils, #microglia, #IL-17 https://t.co/mH5PoQn3FG
nature.com
Nature Medicine - APOE4-dependent neutrophil–microglia interactions drive cognitive impairment in female carriers with Alzheimer’s disease via IL-17F.
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Our new work published today @NatrureMedicine
https://t.co/QzfpAxgKpB The work led by Neta Rosenzweig (@NetaRose11) and Kilian Kleemann (@KilianKleemann). Thanks to our collaborators @EggenBart, @BlurtonJonesLab, @ReisaSperling @weinerlabhms @NowarskiLab
nature.com
Nature Medicine - APOE4-dependent neutrophil–microglia interactions drive cognitive impairment in female carriers with Alzheimer’s disease via IL-17F.
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Another tour de force from @OlegButovsky lab further expanding our understanding of the APOE biology in Alzheimer’s disease. Congrats to the entire team of authors!
Our new work published today @NatrureMedicine
https://t.co/5VMROamUUa… The work led by Neta Rosenzweig (@NetaRose11) and Kilian Kleemann (@KilianKleemann). Thanks to our collaborators @EggenBart, @BlurtonJonesLab, @ReisaSperling @weinerlabhms @NowarskiLab
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Our new work published today @NatrureMedicine
https://t.co/5VMROamUUa… The work led by Neta Rosenzweig (@NetaRose11) and Kilian Kleemann (@KilianKleemann). Thanks to our collaborators @EggenBart, @BlurtonJonesLab, @ReisaSperling @weinerlabhms @NowarskiLab
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Sex-dependent APOE4 neutrophil–microglia interactions drive cognitive impairment in Alzheimer’s disease @NatureMedicine
https://t.co/bjHw0WaLJg
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APOE4 locks microglia in a state of homeostasis, preventing them from clearing amyloid plaque or beckoning astrocytes to help. Deleting APOE4 stirred microglia to dispose of plaques and tau tangles. @BrighamWomens @MayoClinic @hkust
https://t.co/FHdcTTgydW
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@historyinpic66 אתה יודע מה היופי. שגם אם דעתי שונה משלך, אלמד להקשיב ולקבלה. שתזכה להתחדש כל פעם מחדש לטובה🌷 קודם כל לחתור להחזיר את כל השבויים. ולהפוך את רחל אדרי לראש הממשלה🙃
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Dear Harvard Community and Alumni, We, the Israeli students at Harvard College, are reaching out to you during a time of profound grief and urgency. Over the weekend, the community of Kfar Azza, situated right next to the Gaza border, experienced an unimaginable tragedy.
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During this darkest hour, the Sun encourages you to join us in extending a helping hand to the Israeli kibbutzim as they begin their long journey to recovery.
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Dear Friends and Colleagues, A devastating terror shook Israel, targeting kibbutzim. This tragedy deeply saddens us. In response, our Boston community, alongside a local Israeli non-profit organization, has launched a campaign to aid the affected families in their recovery.
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