Mason
@MasonSweatPhD
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Postdoctoral Research fellow investigating the molecular determinants of arrhythmias at Boston Children’s Hospital.
Boston, MA
Joined January 2020
The Sweat lab will be opening at UTSW, the Moss heart Center, in September!. Research topics include AFIB and cellular identity programs. I'm grateful for this opportunity, made possible by support from my family, amazing academic mentors, and the wonderful Moss Cntr. Faculty!.
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RT @MatthiasNahrend: With boost to NIH budget, Senate panel rejects Trump’s plan to slash agency | Science | AAAS
science.org
Republicans on spending committee push back against proposed reorganization and overhead cuts
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RT @junedh_amrute: We discover RUNX1 as a nodal regulator of human cardiac recovery and uncover a druggable epigenetic mechanism in macroph….
biorxiv.org
Despite advances in disease treatment, our understanding of how damaged organs recover and the mechanisms governing this process remain poorly defined. Here, we mapped the transcriptional and...
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RT @glynnis_bann: Excited to share our study out in Circulation identifying the ability of PHF7 to activate cellular reprogramming and rec….
ahajournals.org
BACKGROUND: Direct reprogramming of fibroblasts to cardiomyocytes is a potentially curative strategy for ischemic heart disease. However, current reprogramming strategies require excessive factors...
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RT @rongbin_zheng: I am beyond honored and humbled to receive the Pathway to Independence Award (K99/R00) from the NIH @NHGRI. Deeply grate….
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RT @FengXiao1000: Thrilled to share that I've been awarded @AdditionalVent Single Ventricle Research Fund! This means a lot to me at this s….
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RT @evgenykvon: Our preprint describing the Range Extender element, which is required and sufficient for long-range enhancer activation at….
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RT @AmandaTong2013: New paper alert! @CircAHA @utswheart The first paper from my independent lab! great collaboration with @SChung_Lab @a….
ahajournals.org
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RT @JohannesBacks: Maarten van den Hoogenhof found a specific way to improve cardiac function in RBM20 cardiomyopathy. CAMK2 inhibition mig….
biorxiv.org
Although heart disease can arise from different etiologies, current treatment is not tailored to the different underlying causes but is rather a one-size-fits-all approach. Importantly, not all...
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RT @NatureCVR: Research| Adamopoulos, Rovas et al. show that high regional wall shear stress correlates with fibrosis and left atrium elect….
nature.com
Nature Cardiovascular Research - Adamopoulos, Rovas et al. show that high regional wall shear stress correlates with fibrosis and electrical scarring in the left atrium of patients with atrial...
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RT @CircRes: Methawasin & colleagues found reducing harmful protein clumps in heart muscle cells with a special molecule called antisense o….
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RT @tangming2005: 1/. Creating a figure as a bioinformatician?. 10% making the plot. 90% changing fonts, colors, labels, layout. Let’….
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RT @FarahSheikhLab: 📢 New publication on AAVrh10-based plakophilin-2 gene therapy, showcasing exciting preclinical data supporting the rece….
nature.com
npj Regenerative Medicine - Preclinical efficacy and safety of AAVrh10-based plakophilin-2 gene therapy (LX2020) as a treatment for arrhythmogenic cardiomyopathy
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RT @CircAHA: #ICYMI: Yap and mitotic rounding in cardiomyocytes @jkim @RichGangLi @ShijieLiu_CCHMC @jmartinlab.
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RT @IntMedAbel: Check out our latest publication in @CircAHA .@AHAScience with @LongShengSong1 & @qianshi_uiowa We show how Insulin Recepto….
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RT @njvdn: Our pre-print on in vivo cardiac applications of CRISPR/AAV-mediated precise genome editing is now available! We present two exa….
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RT @njvdn: Scientists trying to plan across the gap between NIH grant application and award activation
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Here we show that in addition to the repressor function of CHD4 as a component of the NuRD complex, it can also act as an activator through genomic recruitment by TBX5. CHD4 recruitment drives the atrial cardiomyocyte gene program and prevents Atrial Fibrillation.
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