Nanashī 🫀 Profile Banner
Nanashī 🫀 Profile
Nanashī 🫀

@The_Nanashi_O

Followers
3,326
Following
846
Media
854
Statuses
20,967

I study ECGs and POCUS

England, United Kingdom
Joined February 2015
Don't wanna be here? Send us removal request.
Explore trending content on Musk Viewer
@The_Nanashi_O
Nanashī 🫀
2 years
@dedoyinajayi You don't apologise to gain forgiveness or feel good. You apologise to make the offended/wronged see that your actions are regretted and you're ready to right wrongs. Whatever response you get, as long as it is not "negative" MUST be "acceptable" to you.
26
286
4K
@The_Nanashi_O
Nanashī 🫀
2 years
@dedoyinajayi You most definitely should not be looking to control the outcome of your apology by expecting/demanding a certain feel-good response. And, "it's OK" is as magnanimous a response as it gets.
6
35
324
@The_Nanashi_O
Nanashī 🫀
2 years
@DrLongissimus Farmer walks into the ED. Activate everything.
1
8
155
@The_Nanashi_O
Nanashī 🫀
1 year
@EM_RESUS Every time!
@The_Nanashi_O
Nanashī 🫀
2 years
@DrLongissimus Farmer walks into the ED. Activate everything.
1
8
155
2
5
129
@The_Nanashi_O
Nanashī 🫀
4 years
@ojayfemi @DGlaucomflecken –7.5 here. Let's just say, it's not been easy. I'm paralysed without my specs.
3
0
117
@The_Nanashi_O
Nanashī 🫀
1 year
@drkeithsiau Respiratory ran out of bed space and there's a history of something remotely gastroenterological from 10 years ago.
3
3
111
@The_Nanashi_O
Nanashī 🫀
2 years
@OGdukeneurosurg Third eye in the wrong place.
1
3
100
@The_Nanashi_O
Nanashī 🫀
11 months
Tweet media one
12
21
84
@The_Nanashi_O
Nanashī 🫀
3 years
30-something hypertensive male with right-sided pleuritic chest pain for 1 week. @smithECGBlog @BrooksWalsh @EM_RESUS @brun_dav @MaruanCarlos @adribaran
Tweet media one
19
11
70
@The_Nanashi_O
Nanashī 🫀
2 years
@shifter_cat @dedoyinajayi Agreed! If the offended responds positively, it is their graciousness that informs it. If the offender doesn't get a positive response, they are at the mercy of the offended. There are nuances to this, but this is the way of offence, forgiveness, and relationships.
1
0
65
@The_Nanashi_O
Nanashī 🫀
11 months
@HandtevyMD @EM_RESUS Annoy, he said, not terrify.
1
0
63
@The_Nanashi_O
Nanashī 🫀
3 years
57-YO known hypertensive presented with tiredness, profuse sweating and headaches. BP 234/137 mmHg, PR 80 bpm. Thoughts? Best picture sent to me. Please bear with me. @smithECGBlog @BrooksWalsh @EM_RESUS @brun_dav @MaruanCarlos @rob_buttner @PacoDardon @ecgrhythms @AslangerE
Tweet media one
17
12
58
@The_Nanashi_O
Nanashī 🫀
11 months
@EM_RESUS "Recorded immediately" should mean that we wait a while and get a repeat because the low-flow state/expected relative acidosis can produce all sorts. If we had to take this one at face value, then the very wide QRS could be any sodium channel poison — ⬆️ K+, ⬇️pH, SCB meds, etc.
0
0
57
@The_Nanashi_O
Nanashī 🫀
9 months
@EcgsOnly Good example of how PVCs can show acute ischaemia better than sinus beats. Through the PVCs, we can see inferior, posterior, and lateral injury. We can also see anterior injury (TWs of PVCs in V3, V4) coupled with a suspiciously flat ST in V1 (sinus) to suggest a pRCA OMI.
1
4
52
@The_Nanashi_O
Nanashī 🫀
1 year
@First10EM @DGlaucomflecken Didn't expect that. 😂
0
0
49
@The_Nanashi_O
Nanashī 🫀
4 years
A 32-year-old primigravida at GA 22 weeks with 2 episodes of sudden fainting spells and spontaneous recovery. Electrolytes are normal. This was all the history I got. @ecgrhythms @ojayfemi @BrooksWalsh @EM_RESUS @smithECGBlog
Tweet media one
12
12
44
@The_Nanashi_O
Nanashī 🫀
1 year
@EM_RESUS Sodium channel blocker toxicity. Take your pick between TCA overdose and hyperK+. Sinus tachycardia and RAD tilts me towards TCA tox.
0
1
45
@The_Nanashi_O
Nanashī 🫀
2 years
@EM_RESUS Knee must be stuck in the chest.
1
0
42
@The_Nanashi_O
Nanashī 🫀
1 year
Tweet media one
19
8
41
@The_Nanashi_O
Nanashī 🫀
2 years
@EM_RESUS SR + qRBBB + Acute proximal LAD OMI pattern, until proven otherwise. Old inferior injury. Dissonance in the ST shift from V2—4 is probably due to high placement of V1, V2.
1
2
42
@The_Nanashi_O
Nanashī 🫀
9 months
@gerben_robbers 1. Kosuge's sign: with acute chest syndrome, TWI in V1 and III 1b. TWI in the chest leads will often involve V1—3 or 4 2. S1Q3T3 3. Tachycardia, typically sinus
2
5
41
@The_Nanashi_O
Nanashī 🫀
3 years
From a colleague: 40/M with retrosternal CP. ECG 1 at t-0 and ECG 2 at t-5 hrs. Initial trops negative. Thoughts? @smithECGBlog @EM_RESUS @BrooksWalsh @PendellM @TahaMD_EM @MaruanCarlos @brun_dav @PacoDardon
Tweet media one
Tweet media two
10
7
36
@The_Nanashi_O
Nanashī 🫀
2 years
49/M, HTN, walks with a c/o poor sleep over the past few weeks. No other symptoms. Without the benefit of the history, would this be interpreted as AMI? @PendellM @BrooksWalsh @smithECGBlog @DidlakeDW @ecgrhythms @MaruanCarlos @brun_dav @adribaran @FloydECGs @TahaMD_EM
Tweet media one
4
7
35
@The_Nanashi_O
Nanashī 🫀
2 years
@RJS_med First ECG was already diagnostic of inferoposterior OMI, in the right context. Second looks more scary, but the first is no less real. Either way, making it a habit to get serial ECGs is simply gold. 👍
1
1
37
@The_Nanashi_O
Nanashī 🫀
2 years
A shared by a colleague: 40/M Obese. Rushed to the ER following a fall with faecal incontinence and altered consciousness, weak pulses, unrecordable BP, SpO² 96%, RBG 13.7 mmol/L. @smithECGBlog @BrooksWalsh @EM_RESUS @AslangerE @DidlakeDW @TahaMD_EM @FloydECGs @PendellM
Tweet media one
12
8
36
@The_Nanashi_O
Nanashī 🫀
2 years
23-yo female with central chest pressure of a few hours duration 24 hours after ingesting marijuana-laced cake. @smithECGBlog @UlhasDr @ecgrhythms @ekgpress @EM_RESUS @BrooksWalsh @brun_dav @MaruanCarlos @TahaMD_EM @syamkumarmd @ECGfan @PacoDardon
Tweet media one
13
12
35
@The_Nanashi_O
Nanashī 🫀
10 months
50s/M with chest discomfort and diaphoresis. #ECG #FOAMed #CardioTwitter
Tweet media one
Tweet media two
9
7
35
@The_Nanashi_O
Nanashī 🫀
11 months
This was hyperK+ 8.0 mmol/L. The TWs in the praecordial leads (not tall) have a narrow base than expected and the PRI is prolonged. Remarkably, the QRS complexes remain quite narrow (86 ms). This was a case of obstructive uropathy.
6
6
33
@The_Nanashi_O
Nanashī 🫀
9 months
@EM_RESUS Transmural injury pattern in the LAD territory. Given likely blunt force trauma to the chest (or maybe even penetrating), the possibilities are: 1. Myocardial contusion 2. Traumatic LAD/LM injury (dissection, rupture, etc) 3. A badly-timed thrombotic LAD OMI. Contrast chest CT.
0
1
31
@The_Nanashi_O
Nanashī 🫀
3 years
@AdityaMandawat Acute calcium gluconate/chloride deficiency.
0
0
32
@The_Nanashi_O
Nanashī 🫀
2 years
@EM_RESUS Acute proximal LAD or, less likely, LM occlusion. Q waves develop within the the first hour in ½ of MIs. The presentation sounds like either LV dysfunction with reduced CO vs papillary rupture with MR and pulmonary oedema. Activate Cath lab but quick echo just to see.
0
1
31
@The_Nanashi_O
Nanashī 🫀
2 years
28-yo female, 3 days post Caesarian delivery, with c/o sudden onset of breathlessness, intermittent palpitations dizziness, and vague chest discomfort. Vitals stable, RR 18, SpO² 98-99% in room air. @BrooksWalsh @smithECGBlog @ecgrhythms @DidlakeDW @gerben_robbers @EM_RESUS
Tweet media one
17
8
30
@The_Nanashi_O
Nanashī 🫀
4 years
75-yo male with no history of cardiovascular disease. PMH is only significant for dyspepsia. What do you make of this ECG? @smithECGBlog @BrooksWalsh @ecgrhythms @EM_RESUS
Tweet media one
10
6
30
@The_Nanashi_O
Nanashī 🫀
9 months
Courtesy @Rodrigue_Ekollo Had trouble figuring this one out because of the machine configuration. NB: First 3 beats in the rhythm strip is reproduced in all 12 leads. #EPeeps #ECG #CardioTwitter @syamkumarmd @MaruanCarlos @Arron_Pearce_ @DidlakeDW @DaveRichley
Tweet media one
9
6
29
@The_Nanashi_O
Nanashī 🫀
2 years
@docnadel @ecgrhythms @BrooksWalsh @smithECGBlog @ECGTalk @SchakrabartiEP @amalmattu @EM_RESUS @EMS12Lead @ECGfan @narrowQRS @Basalus Junctional escape rhythm with retrograde P waves whose repolarisation waves (Ta) — which are of opposite polarity to the PWs — cause a pseudo-ST elevation. We see the reverse in leads with an upright retrograde P wave. Praecordial leads look like old anterior MI.
3
4
30
@The_Nanashi_O
Nanashī 🫀
10 months
@EcgsOnly Pericarditis is a possibi...
1
0
28
@The_Nanashi_O
Nanashī 🫀
1 year
@drkeithsiau Waterhouse-Friderichsen syndrome? Only thing I can tie adrenal failure to septic shock right now.
2
0
29
@The_Nanashi_O
Nanashī 🫀
2 years
@dedoyinajayi Doing this means the ego was never dropped.
1
2
28
@The_Nanashi_O
Nanashī 🫀
10 months
60s ♂️ with this ECG described as "mostly normal". Do you see anything abnormal? 1st ECG — 2½ months prior 2nd ECG (LLs and V leads) — today #ECG #FOAMed #CardioTwitter
Tweet media one
Tweet media two
Tweet media three
10
3
26
@The_Nanashi_O
Nanashī 🫀
2 years
89-yo male with severe peripheral artery disease. No chest pain.
Tweet media one
8
7
26
@The_Nanashi_O
Nanashī 🫀
10 months
Asked about the likely site of origin of the PVCs. What do you think? #FOAMed #ECG #CardioTwitter #EPeeps
Tweet media one
13
5
28
@The_Nanashi_O
Nanashī 🫀
1 year
@EcgsOnly 2:1 AFL/AT with LBBB-type aberrancy. Leads V1 and V2 show both sets of flutter/atrial waves. One just before the peak of the T and another just before the next QRS complex.
Tweet media one
1
2
27
@The_Nanashi_O
Nanashī 🫀
3 years
Tweet media one
8
5
26
@The_Nanashi_O
Nanashī 🫀
1 year
@EcgsOnly Brugada pattern in V1 and V2 and in aVL (extra-praecordial manifestation) + the history = BrS. When's the ICD going in?
3
0
25
@The_Nanashi_O
Nanashī 🫀
3 years
Came across this ECG in a stack. Only medical history available is hypertension. What do we think? @smithECGBlog @BrooksWalsh @brun_dav @MaruanCarlos @ecgrhythms @TahaMD_EM @ekgpress @EM_RESUS
Tweet media one
9
8
25
@The_Nanashi_O
Nanashī 🫀
1 year
@SergioPinski 1. LA/LL lead reversal 2. AFL/AT (sawtooth, so obviously AFL more likely) 3. Complete LBBB 4. Bi-level AV block — 2:1 at the upper level and 3:2 at the lower level with Wenckebach periodicity (giving a cumulative 6:2 AV conduction pattern)
0
0
25
@The_Nanashi_O
Nanashī 🫀
7 months
@ecgandrhythmRoe 4 reasons: 1. RV injury 2. 2:1 AV block — unsure of this from this tracing but it does appear to be present 3. LV injury (combination of 1 and 3) 4. Bezold-Jarisch reflex Culprit here is the proximal RCA.
1
2
23
@The_Nanashi_O
Nanashī 🫀
9 months
Adult male with chest pain. What's wrong with this tracing? List everything you can see. #FOAMed #MedEd #ECG #CardioTwitter
Tweet media one
10
5
23
@The_Nanashi_O
Nanashī 🫀
2 years
Tweet media one
8
5
22
@The_Nanashi_O
Nanashī 🫀
1 year
@EcgsOnly Easily VT. AV dissociation clearly seen in multiple leads. Negative concordance in the praecordial leads. QS waves in anterior and inferior leads make me think scar-mediated VT (remote LAD OMI).
1
1
23
@The_Nanashi_O
Nanashī 🫀
4 years
I came across this ECG belonging to a middle-aged woman without any history beyond "previous chest pain". It was termed "acute myocardial infarction". I was thinking LVA. Please, lend your thoughts. @BrooksWalsh @ecgrhythms @smithECGBlog @PendellM @EM_RESUS
Tweet media one
8
8
20
@The_Nanashi_O
Nanashī 🫀
1 year
32-yo Caucasian/F, asymptomatic. ECG for pre-op evaluation. No hx of CV disease. Only known CVS risk factor is obesity. No family hx of heart disease. @BrooksWalsh @smithECGBlog @ekgpress @EcgOxford @DidlakeDW @RobertHermanMD @ecgrhythms @EM_RESUS @ECGcases @adribaran
Tweet media one
11
7
23
@The_Nanashi_O
Nanashī 🫀
1 year
@OGdukeneurosurg You mean, CAT scan.
0
0
22
@The_Nanashi_O
Nanashī 🫀
1 year
@EcgsOnly A weird de Winter pattern but still unmistakeable. Acute LAD OMI, enough to cause transmural injury (HATWs in V1—3) but not complete, and so subendocardial ischaemia superimposed. Tachycardia portends LV dysfunction with pulmonary oedema vs cardiogenic shock. Prognosis bad.
0
0
22
@The_Nanashi_O
Nanashī 🫀
1 year
Tweet media one
5
9
21
@The_Nanashi_O
Nanashī 🫀
3 months
@smithECGBlog @PendellM We get the best sense of the QRS duration in lead II — a whopping 280 ms just by eyeballing and probably slightly more! Only 2 entities can produce such a superwide QRS: 1. Severe hyperK (itself a Na+ channel blocker) 2. Pharmacologic Na+ channel blocker
0
0
21
@The_Nanashi_O
Nanashī 🫀
10 months
@narrowQRS Looks like two different artefacts mimicking AFL. One is more conspicuous and originates in the left leg (sparing lead I) and the other does a better job of mimicking actual AFL/AT and is affecting every lead but I can still make out a normal P wave in lead I. Needs repeating.
Tweet media one
2
3
20
@The_Nanashi_O
Nanashī 🫀
2 years
@Tellerzee @esther_toluu I stand for too long, I get cramps, I get tired, my back hurts, I lose concentration, and my productivity drops. From then on, my only aim is to get out of the clinic as fast as possible. Learning can and should be less stressful.
1
3
20
@The_Nanashi_O
Nanashī 🫀
1 year
@RobertHermanMD @PMcardioBot @smithECGBlog @PendellM @DidlakeDW @BrooksWalsh @tbouthillet @Vadeboncoeur_Al @ecgrhythms @EM_RESUS Doesn't look like acute OMI to me. V1, V2 are too high. SR with RBBB and RAD (not convincing for LPFB). Abrupt voltage loss after V4 makes me think left-sided pneumothorax, especially with the history. Naturally, I'd get more ECGs, CXR/Chest CT, and echo.
2
1
19
@The_Nanashi_O
Nanashī 🫀
1 year
@EM_RESUS Inferior injury is obvious. HATWs + reciprocal ST-T changes in leads I and aVL. V1 and V2 are too high, so dubious about ST-T changes there to suggest posterior involvement but this is at least an evolving inferior OMI. Do the usuals, then straight to cath.
0
0
20
@The_Nanashi_O
Nanashī 🫀
4 years
@the_beardedsina Insulin – dextrose/glucagon Beta blockers – glucagon Warfarin – vitamin K Cholinomimetics – atropine TCAs – normal saline + sodium bicarbonate Etc
1
6
18
@The_Nanashi_O
Nanashī 🫀
3 years
@EM_RESUS Acute posterolateral wall injury pattern. OMI is most likely. STE + large T waves in I and aVL with reciprocal ST-T changes in II, III, aVF. STD in V1—6, maximal in V3—4. LCx or OM1 culprit for me.
2
0
19
@The_Nanashi_O
Nanashī 🫀
1 year
@Dave_LewMD Hang on a minute.
0
1
18
@The_Nanashi_O
Nanashī 🫀
2 months
@smithECGBlog @willyhfrick OMI with reperfusion. Normal coronaries could mean, if this is type I MI, the thrombus has autolysed and one will need IVUS to recognise the site of plaque rupture. Alternatively, it could be vasospasm or myocardial bridging or any other cause of type II MI.
0
0
18
@The_Nanashi_O
Nanashī 🫀
8 months
@IM_Crit_ @BrooksWalsh , I think I gasped a little after seeing the CT for "chest discomfort". I was expecting a PE.
1
0
18
@The_Nanashi_O
Nanashī 🫀
11 months
@ManualOMedicine
Manual Of Medicine
11 months
A 78 y.o. female is admitted after a fall. Her history raises the possible of syncope. She continues to remain lightheaded. An ECG is obtained. - What's your interpretation ? 👉 Answer and Interpretation: #medtwitter #foamed #ecg #CardioTwitter
Tweet media one
14
12
104
5
3
18
@The_Nanashi_O
Nanashī 🫀
2 years
@EM_RESUS Inferior, posterior (we know, we know), and lateral OMI. Big RCA. Would've quickly moved over V4—6 to the right to r/o RVMI.
1
1
17
@The_Nanashi_O
Nanashī 🫀
4 years
@AngryTiger__ Having to swim through corpse-infested waters to find the corpse of your son murdered extrajudicially by those meant to protect and enforce the law, and then to not have justice. #EndSARS
0
2
16
@The_Nanashi_O
Nanashī 🫀
2 years
@EM_RESUS SR + 2:1 AVB and ventriculophasic arrhythmia + LBBB. Disparity between ST in V1 and V2—4 is weird in LBBB so maybe there's silent (?chest pain) RVMI and PWMI here (and so, pRCA OMI). AVN ischaemia would explain the rhythm disturbance.
0
0
18
@The_Nanashi_O
Nanashī 🫀
2 years
@MaruanCarlos 1. Bradycardia (due to CHB) 1b. CHB by itself with the AV dissociation 2. Injury of the inferior, posterior, and lateral walls. LVEF could be impaired 3. Attenuated STD in V1 suggestive of RVMI as well. RVMI with RV dilation would severely diminish preload
2
2
18
@The_Nanashi_O
Nanashī 🫀
1 year
@smithECGBlog @PendellM ECG 1 = LVH (confirm with echo). ECG 2 = LAD OMI (Cath lab).
1
0
18
@The_Nanashi_O
Nanashī 🫀
1 year
@EcgsOnly Not asystole. ECG machine on? Yes. Patient "plugged in"? No.
0
1
18
@The_Nanashi_O
Nanashī 🫀
11 months
@EM_RESUS Tis but a scratch.
0
0
17
@The_Nanashi_O
Nanashī 🫀
1 year
@JasonWinterECG 1:1 AFL with RBBB aberrancy (phase III block). Shock.
0
0
17
@The_Nanashi_O
Nanashī 🫀
10 months
"Those are not PVCs" was my reply. 1. P waves precede them (best seen in V1—3) 2. Very narrow/rapid initial aspect of the QRS (best seen in V3, V4) PACs with LBBB aberrancy.
@The_Nanashi_O
Nanashī 🫀
10 months
Asked about the likely site of origin of the PVCs. What do you think? #FOAMed #ECG #CardioTwitter #EPeeps
Tweet media one
13
5
28
1
1
16
@The_Nanashi_O
Nanashī 🫀
1 year
@Sports_Fan7 Nice motion (or tremor) artefact from the left leg (since lead I is spared completely).
0
0
16