Sila Ultanir
@SilaUltanir
Followers
130
Following
97
Media
0
Statuses
59
Joined October 2018
We are recruiting for an exciting project on developing novel therapeutic approaches for CDKL5 deficiency disorder. #CDKL5 . Postdoctoral Project Research Scientist @TheCrick
crick.ac.uk
0
0
0
RT @BatemanLab: Great to see this published and thanks to all 42 co-authors! Including @SilaUltanir @jansen_ac @palneurolab @UKTSA @CellsigL.
0
3
0
RT @n_lorem: Sloane is a special case at n-Lorem—she was the first patient to receive an n-Lorem medicine originally designed for someone e….
0
3
0
RT @biorxiv_neursci: CDKL5's role in microtubule-based transport and cognitive function #biorxiv_neursci.
0
5
0
RT @BatemanLab: Don't forget to register for our 23rd April symposium on rare neurodevelopmental disorders with speakers @a_mctague @SilaUl….
eventbrite.co.uk
Tackling challenges in rare neurodevelopmental disorders: an integrated perspective
0
6
0
RT @n_lorem: For n-Lorem’s Chief Operating Officer, Sarah Glass, the mission is personal. Her son Ethan was diagnosed with a nano-rare muta….
0
2
0
RT @MRC_CNDD: We are delighted to announce that registration for our Spring symposium is open. If you are researching or treating rare neur….
eventbrite.co.uk
Tackling challenges in rare neurodevelopmental disorders: an integrated perspective
0
11
0
Recent publication from our lab showing that CDKL2 phosphorylates CDKL5 substrates in brain. Congratulations to Margaux! . @TheCrick
crick.ac.uk
Scientists at the Francis Crick Institute have found a new treatment target for CDKL5 deficiency disorder (CDD), one of the most common types of genetic epilepsy.
0
5
26
Currently reading New Therapeutic Target for Rare Type of Childhood Epilepsy Identified :
cureepilepsy.org
Our research highlights for the first time a CDKL5 target with a link to neuronal excitability. There is evidence that this calcium channel could be involved in other types of epilepsy too.
0
0
3
RT @maxgabgut: 💥Latest preprint on #LRRK2. Fantastic work of @rebecca_morr in the lab with @enricapel and many others. A link between LRRK2….
biorxiv.org
LRRK2 is commonly mutated in Parkinson’s disease and has cell type-specific mechanisms of activation and function. In macrophages, LRRK2 is associated with lysosomes and is activated following...
0
17
0
RT @TheCrick: Researchers at the Crick, @UCL and @MSDintheUK have identified a potential treatment target for a rare genetic type of childh….
crick.ac.uk
Researchers at the Francis Crick Institute, UCL and MSD have identified a potential treatment target for a genetic type of epilepsy.
0
10
0
RT @VivianLiLab: 📣 Postdoc position opening in the Li lab at the Crick! . We are looking for a highly motivated postdoc to join our vibrant….
0
59
0
RT @TheCrick: We're looking for clinicians who are passionate about research to join our 3-year fully funded clinical PhD programme 🔬🩺.Proj….
crick.ac.uk
The Crick's clinical PhD programme.
0
77
0
Congratulations to Simo, Guillaume and all authors for this fantastic paper. The master energy homeostasis regulator PGC-1α exhibits an mRNA nuclear export function .
pubmed.ncbi.nlm.nih.gov
PGC-1α plays a central role in maintaining mitochondrial and energy metabolism homeostasis, linking external stimuli to transcriptional co-activation of genes involved in adaptive and age-related...
0
1
4
RT @BatemanLab: We have an RA post available to work in our new mTOR Pathway Diseases node, part of the NIHR/MRC UK Rare Disease Research P….
0
20
0
RT @NicoMarichal: Many thanks to our speakers today @jbneuro & @SilaUltanir Lot about Synapses, excitation and inhibition durring brain de….
0
4
0
We have an exciting postdoctoral fellowship opportunity. A collaborative project with Flor Iacaruso's lab at the Crick. Deadline is June 24th. @TheCrick
0
2
9
RT @CDKL5_IFCR: If you are near Athens, Ohio go check out this beautiful public art raising #CDKL5Awareness! Thank you to Team Rooting for….
0
3
0
RT @CDKL5_IFCR: Will you help us Move CDKL5 Forward?. We have BIG goals for CDKL5 Awareness Month and we need all the help we can get!. 🌟Do….
0
1
0