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Ben Prosser Profile
Ben Prosser

@Prosser_Lab

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2K
Following
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406

Assoc. Prof @PennMedicine | Rare-disease dad. Trying to fix hearts AND brains. Myocytes, Microtubules, Heart Failure | ASOs, RNA, NeuroDevelopmental Disorders.

Philadelphia
Joined March 2020
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@Prosser_Lab
Ben Prosser
3 years
What a privilege to have the team and opportunity to try and help my daughter and those like her live healthier, fuller lives. We will be a relentless force for good against #RareDisease. @IngoHelbig @lab_davidson @ChildrensPhila @PennMedicine
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@ZEISS_Group
ZEISS Group
1 year
Prof. Benjamin L. Prosser and his team at the @Prosser_Lab at @PennMedicine are researching the heart on a molecular level. Learn more about how his personal story motivated him to work on transformative cardiac and gene research. Visit https://t.co/IeOEPY0z95 #Health
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@CircRes
Circulation Research
1 year
@LucieCarrier84 lab provide evidence that targeting #tubulin #tyrosination could be a novel approach for improving heart function in patients with #HCM. Learn more at https://t.co/6FzAYF4rXO @Prosser_Lab @mcaporizzo @CyxChristina @dirkwestermann @LucasBacmeister
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@FiladelfiaGene1
Rikke S. Møller
1 year
This study, conducted in close collaboration with @curestxbp1, aimed to identify the rate & causes of early mortality in #STXBP1-related disorders. These results are important for family & prognostic counselling💜 @FrancescaFuria_ @Elegardella 👏 https://t.co/8WqyAWJqTg
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link.springer.com
Neurological Sciences - Pathogenic variants in STXBP1 cause a spectrum of disorders mainly consisting of developmental and epileptic encephalopathy (DEE), often featuring drug-resistant epilepsy....
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@Prosser_Lab
Ben Prosser
1 year
$$$ for #RareDisease research! RFAs for 35 different rare disorders through the @ODC_UPenn, including 2 x $70K grants available to @curestxbp1 courtesy of Lulu's Crew :) Info and app link here: https://t.co/agZMdLsrGr
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@ScienceTM
Science Translational Medicine
1 year
Small molecules that inhibit vasohibins and alpha-tubulin #detyrosination can promote myocardial relaxation and counteract heart muscle cell stiffness in a rat model of #HeartFailure with preserved ejection fraction. @MarguliesLab @Prosser https://t.co/pfvLktEr6T
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@Prosser_Lab
Ben Prosser
1 year
Want to know more about what ENDD is doing for rare 🧠disorders like @cureSYNGAP1 and @curestxbp1? Updated website here! https://t.co/LwbLZe0v6V @ChildrensPhila @PennMedicine @lab_davidson @IngoHelbig @EAHellerPhD
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@Prosser_Lab
Ben Prosser
1 year
Next big step - determine chronic efficacy (and safety!) with sustained treatment to prevent progression of HFpEF (and other forms of HF). Awesome effort from @tweetineaton @MCaporizzo and Ben Lee.
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@Prosser_Lab
Ben Prosser
1 year
In vivo, overnight VASHi was well tolerated, and improved ventricular relaxation and diastolic function in advanced HFpEF. (opinion - that's pretty cool for just targeting a microtubule PTM!) 4/5
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@Prosser_Lab
Ben Prosser
1 year
ZSF1-Obese rats capture many features of human HFpEF. Ex vivo, we found that VASH inhibition (VASHi) lowered cardiomyocyte stiffness and improved relaxation in HFpEF cardiomyocytes. 3/5
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@Prosser_Lab
Ben Prosser
1 year
Microtubule detyrosination (dTyr) is increased in human heart failure, and contributes to cardiac stiffening. We tested a new, potent small molecule inhibitor of the detyrosinase (VASH) to see if it could lower stiffness and improve function in a rat model of HFpEF. 2/5
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@Prosser_Lab
Ben Prosser
1 year
Can we "drug" microtubule modifications to lower cardiac stiffness and improve function in #HFpEF? Out now in @ScienceTM , w/ long time collabs in @MarguliesLab 1/5 https://t.co/gtcpuo7dFI
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@Heart4Carolina
Leslie K. 👩🏾‍🔬
1 year
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@KehatLab
Kehat Lab
1 year
Check out our new paper @jclinicalinvest by the great @Ramihdd and @shkedio. We show that the protein RPSA is required for ribosome localization, localized translation, and the maintenance of the sarcomere.
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@Prosser_Lab
Ben Prosser
2 years
Happy birthday Lu! 6 years - and now standing by yourself! I have too many feelings to tweet, so I'll just say THANK YOU to everyone who has supported Lucy, or who is fighting to improve the lives of those with #raredisease. Keep going.
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@Prosser_Lab
Ben Prosser
2 years
Keep going Brett.
@UFDTech
Brett @ UFD Tech
2 years
Someone asked me about the scratches I had on my faces during a recent video. Living life trying to @cureSYNGAP1 isn't always easy, but Emmitt's always worth it.
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@Prosser_Lab
Ben Prosser
2 years
Don't miss the premier conference for fundamental and translational cardiac research! Program in link, still with open talk slots to be selected from abstracts. #CardioTwitter @Rongtian2 @KimHellgren @Heart4Carolina https://t.co/3aUr2tpXkl
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@Prosser_Lab
Ben Prosser
2 years
These are BEAUTIFUL 👏tissue mechanics in living myocardial slices by the Caporizzo Lab. They define (with precision) how targeting microtubules can improve cardiac function by facilitating relaxation and early diastolic filling.
@MCaporizzo
Matt Caporizzo
2 years
Excited about our latest work in @JMCCPlus showing colchicine improves diastolic performance in myocardial slices. Congrats @EmmaleighHancoc and thanks to Brad Palmer, @IonOptix , @UVMLarnerMed , and @uvmcvri for making this possible! https://t.co/wb2o6q8wPT
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@Prosser_Lab
Ben Prosser
2 years
Happy #RareDiseaseDay! Lucy, 5, #STXBP1, working HARD on that first word (Hiiiiiii!) and independent steps. Effortlessly bringing all the sunshine ☀️
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@KimHellgren
Kim Hellgren
2 years
Deadline alert! March 17th is the last day to submit your abstract for oral presentation consideration at the Gordon Research Conference on Cardiac Regulatory Mechanisms this June. Don't miss your chance to showcase your research! #CardiacScience #GRC #GRS https://t.co/61Q7XbVJHd
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@spirochrome
Spirochrome
2 years
Rat cardiomyocyte stained with SiR-actin (magenta) & hoechst (cyan) imaged at high frame rate showing nucleus strain during contraction! Don't miss your chance to read the amazing associated preprint "Microtubule forces drive nuclear damage in LMNA cardiomyopathy" from
@Prosser_Lab
Ben Prosser
2 years
@AmiadDaria developed a novel assay to measure how cytoskeletal strain is transferred to the nucleus during the contraction and relaxation of the heart muscle cell. (🧵2/5)
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