Valery Krizhanovsky Profile
Valery Krizhanovsky

@KrizhanovskyLab

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Scientist at the Weizmann Institute. Senescence and Aging research

Rehovot
Joined September 2021
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@Nadav_Eyal
נדב איל Nadav Eyal
5 days
A few weeks ago, I heard the Philharmonic perform in New York, and outside there was a demonstration. It was after the ceasefire, and the people there weren’t protesting Gaza. The signs we saw weren’t about Gaza at all. They were about Israel - about the existence of Israel.
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@IgorUlitsky
Igor Ulitsky
11 days
"It's just a temporary space until your lab is renovated", Weizmann Institute version. Rebuilding better, and doing so fast. (Photo and tagline: Ziv Shulman)
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@KrizhanovskyLab
Valery Krizhanovsky
14 days
WIS rocks!!!
@WeizmannScience
Weizmann Institute
15 days
We're proud to share that the Weizmann Institute of Science has been ranked 6th in the world in the 2025 @cwtsleiden Ranking - a jump of four places from the 2024 ranking and our highest position since the ranking was introduced some two decades ago >> https://t.co/YNbQnHGe0S
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@KrizhanovskyLab
Valery Krizhanovsky
1 month
It would be a pleasure to participate. @CherfilsJulien Thank you for the invitation
@CherfilsJulien
Julien Cherfils
1 month
📢 We organize a Workshop “Senescence Immune Surveillance: Implications in Cancer and Aging” 🗓 October 22, 2025 – Le Galet Auditorium, Pasteur Hospital Leading experts (Institut Pasteur, Weizmann Institute, Karolinska Institutet…) and Free entry! Don’t miss it!
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@NatureAging
Nature Aging
3 months
🎉Immune aging focus 🎉 @JuliaMMagdalena and @KrizhanovskyLab explore immune surveillance of senescent cells, including mechanisms of clearance, evasion and paracrine senescence, as well as therapeutic strategies https://t.co/2cWxb8evpN
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nature.com
Nature Aging - Majewska and Krizhanovsky discuss the interactions of innate and adaptive immune cells with senescent cells, including mechanisms of clearance, evasion and paracrine senescence, as...
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@KrizhanovskyLab
Valery Krizhanovsky
3 months
Thank you for featuring our review
@LabWaggoner
Waggoner Lab
3 months
Review @NatureAging @JuliaMMagdalena @KrizhanovskyLab Immune surveillance of senescent cells in aging and disease https://t.co/980jc4eTXG
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@IgorUlitsky
Igor Ulitsky
4 months
How we’re rebuilding the Weizmann Institute — and our hopes for a better future
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nature.com
Nature - Scientific research is at the heart of Israel’s success and has worldwide benefits. Our work will continue despite adversity.
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@LabGeiger
Tami Geiger (lab)
5 months
It took me more than 10 days before I could enter Ullman and climb to the burnt lab. Heartbreaking. Thankful to @WeizmannScience for helping us get up on our feet and look forward.
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@KrizhanovskyLab
Valery Krizhanovsky
5 months
Nothing can break us!
@UriAlonWeizmann
Uri Alon
5 months
When we came to see our destroyed labs hit by an Iranian missile, we found that a piano had survived @WeizmannScience
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@KrizhanovskyLab
Valery Krizhanovsky
9 months
Our new website is up and running!!! https://t.co/l1Rka7U5P9
weizmann.ac.il
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@KrizhanovskyLab
Valery Krizhanovsky
10 months
Very interesting study!
@ScienceMagazine
Science Magazine
10 months
New work in @ScienceTM connects immunosenescence and the decline of the thymus during aging with a drop in the ligand RANKL—and shows RANKL treatment restores T cell production and rejuvenates immune responses in aged mice. https://t.co/KhFjz67Ojf
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@KrizhanovskyLab
Valery Krizhanovsky
1 year
One very ineresting view on aging clocks and what they can or cannot tell us. 👇 https://t.co/Fd5cEMcBpT
nature.com
Nature Medicine - Biological aging clocks are flawed concepts in understanding disease.
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@ICSA_senescence
International Cell Senescence Association
1 year
Save the date! @marc_dema announced that the next ICSA meeting will be held in Rome, marking the 10th anniversary of the ICSA meetings. Get ready for #ICSARome2025! 🇮🇹
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@KrizhanovskyLab
Valery Krizhanovsky
1 year
Congrats Nurit. In this stidy we show that p21 might be master regulator of cellular microenvironment with a critical role in fibrosis. Thanks to all the coauthors.
@embojournal
The EMBO Journal
1 year
What is the contribution of p21-positive senescent cells to tissue fibrosis? @KrizhanovskyLab @WeizmannScience show that p21 controls not only cell cycle progression but also promotes expression of ECM components and immune invasion https://t.co/dxZvvYNdxr
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@Tzahore
Eldad Tzahor
1 year
Incredible program to apply to
@PostdocIsrael
Postdoc in Israel
1 year
Attention prospective postdocs! The Azrieli International Postdoctoral Fellowship for Research at Israeli Universities in STEM, Humanities, and Social Sciences is now open for applications! You can apply today for the 2025–26 cohort. The Azrieli International Postdoctoral
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@amitmechaging
Amit Agrawal
1 year
Immunotherapy a key to fight aging? We break down our recent study in this animation! 🧪 Immune checkpoint anti-PD-L1 can enhance the immune system to remove senescent cells & ameliorate age-related phenotypes! https://t.co/KIdDoxwgRF… @JuliaMMagdalena @KrizhanovskyLab
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@KrizhanovskyLab
Valery Krizhanovsky
1 year
The business side of academic publishers... The question is what would be a better model?
@MushtaqBilalPhD
Mushtaq Bilal, PhD
1 year
Major academic publishers' revenue and what they pay authors and reviewers: Revenue: Elsevier: $3.9 billion Springer Nature: $2 billion Taylor & Francis: $800M Wiley: $1.8 billion They pay: Academic authors: $0 Peer reviewers: $0
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@NatureCellBio
Nature Cell Biology
1 year
📑: @JuliaMMagdalena @amitmechaging et al fr/@KrizhanovskyLab show that p16-expressing senescent cells enhance the stability of the immune checkpoint PD-L1, by downregulating its proteasomal degradation to accumulate in ageing and chronic inflammation. https://t.co/rbqPzfKSiM
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nature.com
Nature Cell Biology - Majewska et al. show that p16-expressing senescent cells enhance the stability of the immune checkpoint PD-L1 by downregulating its proteasome-mediated ubiquitin-dependent...
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@JuliaMMagdalena
Julia Magdalena Majewska
1 year
We show that p16 expressing #SenescentCells enhance PDL1 stability, by downregulating its ubiquitin-dependent proteasomal degradation to accumulate in #Aging and chronic inflammation. Anti-PDL1 activates an immune response to eliminate senescent cells and decrease inflammaging.
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