We're proud to share that the Weizmann Institute of Science has been ranked 6th in the world in the 2025 @cwtsleiden Ranking - a jump of four places from the 2024 ranking and our highest position since the ranking was introduced some two decades ago >> https://t.co/YNbQnHGe0S

Highly recommended lab and a great friend 👇

I tried to describe my path from basic science to startups, from academia to industry and how to balance between the two efforts. Sometimes fun, often hard but i don't regret doing it. open link:

My lab at @WeizmannScience was completely demolished by the Iranian missile a few days ago. This is hard and sad as nothing was left to save. We study heart regeneration and regenerative medicine. As such, we will regenerate, regrow and rebuild. The one thing that wasn't gone is

Cardiac regeneration, @WeizmannScience #8400, my entrepreneurial efforts and more... in the Israeli Trailblazers Podcast with Jennifer Weissmann https://t.co/UuvMLN9a0M

While hard to see myself here. It was fun to chat with Jennifer about science and entrepreneurial paths I have taken in recent years and on @WeizmannScience and #8400

Our paper plus the cover in @CellSystemsCP on cardiac fibrosis. A novel insight on these pathologies with some therapeutic solutions.

Are you passionate about live-imaging microscopy? Do you have a special talent for working with iPSCs? Or are you simply a zebrafish aficionado? 🐟 We have 2 funded postdoctoral positions in our lab ( https://t.co/0PwWpG37zU). Join us and apply now! Please spread the word 🙏

RETWEET if you stand with President Zelensky!

The story of our collaborative cardiac fibrosis paper with @UriAlonWeizmann, @MiyaraShoval, @MiriAdler and many more. https://t.co/BlbkYPqsoE

The story of our study:

Last but not least, we thank @EladBassat and Jingkui Wang from the lab of @Tanaxolotl at @IMPvienna 🦾, Andrea Baehr, and Christian Kupatt @TU_Muenchen. Special thanks to the amazing Dr. Avi Mayo from the @UriAlonWeizmann team & Danielle Kimchi for original illustrations. End.

Huge thanks to Daniel Haußler and Achim Kruger @TU_Muenchen, and the amazing @roramirezf94 and @JulioSaezRod @saezlab @UniHeidelberg @emblebi, and Tatjana Dorn and Alessandra Moretti from DZHK (13/)

Hanna Bueno-Levy, Rachel Sarig from @Tzahor_Lab, Tali Shalit, Michael Gershovits and Eviatar Weizman from @WeizmannScience INCPM🦾🦾🦾 (12/)

This work was made possible thanks to our amazing collaborators from around the world. Thank you to our lab members @JacobElkahal, Kfir Umansky @YalinDivinsky, @genzelinakh, @abingtonSA, @LinglingZhang12, @DavidKain17, Daria Lendengolts, @ZacharyPetrover (11/)

In vivo, we demonstrate that inhibition of TIMP1 following myocardial infarction reduces myofibroblast proliferation and subsequent fibrosis. (10/)

We demonstrate that TIMP1 acts as a growth factor for cardiac myofibroblasts derived from either mouse or non-human-primate hearts. (9/)

Using scRNAseq and NicheNet analyses we identified TIMP1 as a key cardiac myofibroblast autocrine growth factor following myocardial infarction (8/)

Our theoretical model revealed a vulnerability of ❄️cold fibrosis: the myofibroblast autocrine growth factor loop. (7/)

We show that❄️cold fibrosis following myocardial infarction is conserved in mice, pigs, and humans (6/)