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Rohatgi Lab Profile
Rohatgi Lab

@RohatgiLab

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Official account for the Rohatgi Lab @StanfordMed. Studying signaling pathways in development, disease and homeostasis.

Stanford, CA
Joined September 2017
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@RohatgiLab
Rohatgi Lab
4 years
Post-doc and research assistant positions are available in the Rohatgi lab in the areas signal transduction, lipid signaling/metabolism, primary cilia and biomolecular condensates. See detailed descriptions at https://t.co/ADDIA1iGlB jobRxiv 22119 Please RT
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rohatgilab.stanford.edu
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@RohatgiLab
Rohatgi Lab
4 years
Check out this new pre-print from @pawelbiologist's lab!
@pawelbiologist
Pawel Niewiadomski
4 years
Proud to present our latest preprint "The exocyst complex and intracellular vesicles mediate soluble protein trafficking to the primary cilium" by @Sylwia13042567 et al. in collaboration with @RohatgiLab https://t.co/XfmEwlhxnt #cilia #hedgehog
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@JennKong07
Jennifer Kong, PhD
4 years
Huge congratulations to ⁦@schmidt_lab — a 2021 Forbeck Fellow⁩!!!! 🎉🎉🎉🎉🎉🎉
forbeckfoundation.org
The Forbeck Cancer Forums accelerate progress in the fight against cancer by fostering a results driven exchange of ideas.
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@JennKong07
Jennifer Kong, PhD
4 years
Yay! Our pre-print is finally out on BioRxiv! I am thrilled to finally share chapter 3 of our MMM saga: the mystery of MOSMO! Gene-teratogen interactions influence the penetrance of birth defects by altering Hedgehog signaling strength
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biorxiv.org
Birth defects result from interactions between genetic and environmental factors, but the mechanisms remain poorly understood. We find that mutations and teratogens interact in predictable ways to...
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@nchembio
Nature Chemical Biology
5 years
@RohatgiLab, Arun Radhakrishnan, & Christian Siebold review how the Hedgehog (Hh) receptor PTCH1 uses its transporter-like function to inhibit the GPCR SMO, with cholesterol as a ligand for SMO to effect downstream signaling.
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@JennKong07
Jennifer Kong, PhD
5 years
Woohooo!!! 🥳🎉 Our paper just went online @Dev_Cell! The major fundamental question we sought to address was, what are the mechanisms that regulate a receiving cell’s sensitivity to extracellular morphogens? 🧵🧵🧵 https://t.co/u0XbqVcZ7D
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@RohatgiLab
Rohatgi Lab
5 years
Congratulations Maia!
@StanfordBiochem
Stanford Biochemistry
5 years
Maia Kinnebrew has received the 2019-20 Stanford Biosciences Excellence in Service to Graduate Students award! This award recognizes students who devote a large amount of time trying to improve the quality of the Stanford graduate student experience. Congratulations, Maia!
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@RohatgiLab
Rohatgi Lab
5 years
We welcome feedback on our characterization of a novel membrane-tethered ubiquitination pathway critical for the proper patterning of multiple tissues during development. In collaboration with the labs of Cecilia Lo, Teresa Gunn, and L. Aravind. https://t.co/9AtQ9SqyEO
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biorxiv.org
The etiology of congenital heart defects (CHDs), amongst the most common human birth defects, is poorly understood partly because of its complex genetic architecture. Here we show that two genes...
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@StanfordBiochem
Stanford Biochemistry
5 years
Check out this article by Ramin Dubey of the Rohatgi Lab and collaborators just published in eLife! "R-spondins engage heparan sulfate proteoglycans to potentiate WNT signaling"
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elifesciences.org
Heparan Sulfate Proteoglycans function as receptors for the R-spondins, a family of growth factors that amplify the strength of WNT signaling during development and in adult stem cells.
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@RohatgiLab
Rohatgi Lab
6 years
Congrats to Ramin Dubey on his work linking lipid droplets to hydrophobic drug activation: https://t.co/1XwlLIbHMI. We are grateful to our our collaborators and to @OlzmannLab for their News and Views on "enzymatically regulated phase partitioning":
nature.com
Nature Chemical Biology - Lasonolide A is hydrolyzed into a cytotoxic metabolite by a lipid droplet-associated orphan serine hydrolase, showing that lipid droplets can promote drug toxicity by both...
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@RohatgiLab
Rohatgi Lab
6 years
Membrane cholesterol accessibility as the messenger that communicates the hedgehog signal between patched and smoothened. Collaborative work with Arun Radhakrishnan's lab: https://t.co/l2DZ3dycB7 #gpcr #cilia #lipids #signaling #developmentalbiology #crispr
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elifesciences.org
The patched hedgehog receptor inhibits the transmembrane transducer smoothened by reducing the accessibility of cholesterol locally at the membrane of the primary cilium.
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@RohatgiLab
Rohatgi Lab
6 years
Broder's new paper presents a rigorous approach to test the connection between phase separation and function. Distinguished by the use of compositional mutagenesis to tune phase properties linked to hard biochemical readouts. https://t.co/VPl7I3tT3j
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@BroderSchmidt
Broder Schmidt
6 years
Shorter title, new in vitro data, updated single-cell splicing reporter (available @Addgene): our work on TDP43 phase separation and splicing function is now online! https://t.co/ELNY1Me8F4 @RohatgiLab @StanfordBiochem
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@RohatgiLab
Rohatgi Lab
6 years
Nice collaboration with the Siebold lab reveals the deep evolutionary connection between Hedgehog signaling and cholesterol transport-- congratulations to Maia Kinnebrew. Cholesterol is both a substrate and (when coupled to a protein ligand) an inhibitor of Patched!
@nchembio
Nature Chemical Biology
6 years
Our new paper from @RohatgiLab and Christian Siebold’s group at @NDMOxford used structural and MD analysis to show that cholesterol inhibits Patched by inserting into the extracellular domain. Readable link at https://t.co/AR0H6s8TEn.
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@RohatgiLab
Rohatgi Lab
6 years
A review from our own Jenn Kong on biochemical mechanisms of Hedgehog signaling, written with Christian Siebold, is out in Development-- includes an analysis of the many patched and smoothened structures. @Dev_journal
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@StanfordBiochem
Stanford Biochemistry
7 years
The NIH has awarded Jennifer Kong of the @RohatgiLab the K99/R99 Pathway to Independence Award! This award aims to help outstanding postdoctoral researchers complete training and transition to independent, tenure-track or equivalent faculty positions. Congratulations, Jenn!
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