Daisuke Nakada
@NakadaLab
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The Nakada lab at Baylor College of Medicine. We study hematopoietic stem cells, leukemia, and metabolism.
Joined June 2021
Happy to share that our new paper, led by a former postdoc Xiangguo Shi, reporting that the inhibition of guanosine metabolism sensitizes AML to menin inhibition was featured by the editors of Nature Communications. https://t.co/h308MyiTsK
https://t.co/YbEDaeQiLN
nature.com
On this page we provide a snapshot of some of the most exciting works recently published in Nature Communications in cancer research. We cover all aspects of ...
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Delighted to share my research group's latest study, published today @Nature 👉 https://t.co/C3BF2Wnyw9 where we trace the life histories of blood cancer development in patients @sangerinstitute @SCICambridge @Cambridge_Uni
The mutations that cause adult blood cancer can occur as early as childhood or even in the womb 🩸 #BloodCancer #Genomics Read the full story ➡️ https://t.co/IpZrgI8S1q
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Entered my first academic lab in 1978. Fond memories of >40 years in science with great colleagues at public institutions in Kenya, the US and in my country, the Netherlands. Excited to step into the pharma world as head of research/early development at Roche, Basel in March
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Congratulations to CRI Director @SJMorrison_ on receiving the 2022 @ISSCR Public Service Award for his outstanding contributions in public service to the fields of stem cell research and regenerative medicine! https://t.co/WWqCU1EEIm
cri.utsw.edu
The International Society for Stem Cell Research (ISSCR) is honoring Sean J. Morrison, Ph.D., Director of Children’s Medical Center Research Institute at UT Southwestern (CRI), USA, with the 2022...
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https://t.co/ESkpQrnI4Q Product of a fun collaboration with @chasz43 and his team. SEPHS2, a regulator of selenoprotein synthesis, is upregulated in AML by a super enhancer. AML is addicted to selenium for selenoprotein production to detoxify ROS.
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In this new study, we show that NAD homeostasis governed by NMNAT1 is critical for AML stem cells and suppressing this mechanism renders AML sensitive to venetoclax.
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