Ming Li
@MingLiLab
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Member, Memorial Sloan Kettering Cancer Center Views expressed are not affiliated with my employer.
New York, NY
Joined July 2011
High rate ATP production accounts for the Warburg Effect, perhaps in support of energy-demanding processes such as PI3K signaling in effector T cells.
Excited to share our new paper led by @mattabolite in collaboration with Saharon Rosset showing that the Warburg Effect is the result of faster ATP production by glycolysis than respiration. A š§µ. 1/n
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Congrats to Andrea ā¦@SchietingerLabā© for the very well deserved ā¦@CancerResearchā© Frederick W. Alt Award! Brilliant science for cancer cure!!
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RT @CellCellPress: Now online! Dynamic allostery drives autocrine and paracrine TGF-Ī² signaling
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Glad to share our review article @AnnualReviews on tissue-resident innate and adaptive lymphocytes in cancer. Defining their identities and sensing mechanisms shall guide targeting such prewired and priming-dependent defense responses for cancer therapies.
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RT @CancerResearch: 28 cancers are now treatable with immunotherapy, thanks to the work of the Cancer Research Institute. Explore our Impacā¦.
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Thanks to @MSKCancerCenter for covering it!.
MSK's Dr. Ming Li (@MingLiLab) discusses how his lab is working to harness the power of the immune system against #cancer ā including a new paper just out in @ImmunityCP.
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Deeply missing Albert. He is such as an exemplary scholar.
Albert Bendelac (1956-2023)
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Reprogram tumor-associated macrophages to outcompete endothelial progenitor cells and suppress tumor neoangiogenesis. Excited to share our new study @ImmunityCP by brilliant @miahdo1 , Wei Shi, and @CancerResearch fellow @LiangliangJi and collaborators!
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Growing bigger had a survival advantage for non-minimal cells and not growing bigger had a survival advantage for minimal cells.
A mind-blowing paper has come out today in @Nature . In 2016, JC Venter Institute scientists trimmed a bacterial genome to its barest minimum required for life to synthesize what they called a "minimal genome" (. Today, a group of scientists from
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RT @dana_peer: Excited to introduce scKINETICS, an algorithm for joint inference of gene regulatory mechanism and cellular velocity, co-deā¦.
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Reprogram tumor-associated macrophages to outmatch cancer cell competition. Excited to share our new study @Nature by brilliant @CancerResearch fellow @XianZhang2016 and collaborators!
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RT @Joshilabyale: Our lab's latest paper on peripheral tolerance mechanisms in skin is out now in @Nature. Congrats to @martinadamo for leā¦.
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Celebrating cancer immunology and immunotherapy discoveries supported by #CancerResearch (70 years and going strong) and other agencies to create a world #Immune2Cancer
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RT @CellPressNews: Check out this perspective on nutrient acquisition and metabolism control of macrophage responses in tumor, a collaboratā¦.
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Glad to share a perspective on nutrient acquisition and metabolism control of macrophage responses in tumor @ImmunityCP with @CancerResearch fellows @XianZhang2016 @LiangliangJi
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Glad to share a holistic immuno-oncology perspective through the lens of #TGFb @NatRevImmunol with implication for preclinical model choice for drug testing
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RT @ImmunityCP: Online now: Tumor-associated macrophages expressing the transcription factor IRF8 promote TĀ cell exhaustion in cancer httpā¦.
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RT @MSKCancerCenter: New in @ImmunityCP: a team led by the @MingLiLab at MSK reports a unique function for tumor-associated macrophages (TAā¦.
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Tumor-associated macrophages present cancer cell antigen and drive cytotoxic T cell exhaustion. Check out our story from brilliant Briana Nixon, @dkuo @arihakimi and collaborators
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