💪🏽🩸🅾🚑Are you interested in Low Titer O Whole Blood (LTOWB) within trauma resuscitation? . Great conversation with my friend, Lieutenant William Bullock @SouthTexasBlood discussing the LTOWB Hero in Arms Program. Listen to the entire conversation here 👉🏽

Blooducation Baristas - Transfusion Safety and Donor Covid-19 Infection

Blooducation Baristas - "How I became the Blood Bank Guy"

RT @impactlifeblood: Did you know that Dr. Elizabeth Blackwell was the first woman to earn an M.D. in the United States? Dr. Blackwell pave….

SAVE THE DATE!. Join Us for the Next Blooducation Baristas Event! ☕🩸. I’m thrilled to welcome Dr. Joe Chaffin, @bloodbankguy for an exclusive conversation on .February 6 at 1:15 PM CT! . As a master educator, Dr. Chaffin has had an extraordinary impact on the Blood Banking and

Blooducation Baristas - Navigating Hyperhemolysis Syndrome with Dr. Adkins

#FanArt by Shoshana.Age 10.@aCookieGod

Schedule it now! Our next Blooducation Baristas podcast episode featuring @path_brian, discussing the complex topic of hyperhemolysis syndrome. 📅 Date: Friday, January 17, 2025.⏰ Time: 9:30 AM CT.📍 Platform: Live on LinkedIn, X and Youtube. Don't miss this opportunity to

Prehospital Transfusion in Wisconsin

@threadreaderapp unroll.

RT @BloodScientist: #MorphologyMonday. "Hi," said the person on the phone. "Can you please take a look at the film for this patient urgentl….

Effects of bacterial elements (i.e. endotoxin) can lead to cytokine activation resulting in systemic vasodilation (hypotension), myocardial pump and multiorgan failure with FEVER. Remember that this looks different then low output shock (hypovolemic and cardiogenic shock) which.

Finally, we discussed concepts of shock which is systemic hypoperfusion either due to reduction in cardiac output or effective circulating blood volume. #SepticShock results from a localized infection into the bloodstream. What is the main outcome of cytokine activation?.

Venous emboli that originate from the deep leg veins end up in the pulmonary vasculature, called pulmonary embolisms. If they occlude 60% of the pulmonary circulation, it can lead to sudden death from R heart failure. 😱Incredible saddle embolectomy video👉🏽.

There are many fates of a thrombus including dissolution, organization, and propagation. Embolization may also occur which is detached and carried by the blood to a distant site. ⁉When a thrombus from the deep legs embolizes, which circulation does it lodge?.

Mutations in Factor V gene are most common. Leiden mutation cannot be inactivated + resistant to anticoagulant effects of activated protein C. First discovered in the Netherlands, check out the original article and author @voobergj 👉🏽.

Hypercoagulable disorders predispose individuals to thrombosis and can be divided into primary and secondary. Which of the following is the most common inherited cause of hypercoagulability?.

A true thrombus that forms during life is attached to the endothelial lining and have microscopic alternating layers of platelets mixed w/ fibrin, and RBCS which appear lighter and darker layers called #LinesofZahn. Here's a gorgeous case 👉🏽

#Virchow described that there are 3 factors that increase the risk of thrombosis which include 1) Endothelial injury 2) Abnormal blood flow & 3) Hypercoagulability .⁉How do you tell the difference between a clot that develops in vivo vs post mortem?.

Chronic congestion usually occurs in the setting of right-side heart failure & leads to congestion of the central veins. Because of the status of deoxygenated blood (zone 3), centrilobular hepatocytes first become degenerated --> necrotic #nutmegliver.

Congestion is a passive process due to decreased outflow and can be acute or chronic, localized or systemic. Two key sights of congestion are the 🫁lung and the liver. ⁉When chronic congestion occurs in the liver, which hepatocytes are most affected?.