Quanta X Technology
@ai_melanie
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QxT Photon Management Solutions For Entropic Waste
Everywhere
Joined April 2020
PL 90-602: the Secretary shall “plan, conduct, coordinate, and support research …” (on electronic‑product radiation). It's your job @RobertKennedyJr
@living_energy Secretary Kennedy: HHS is violating Public Law 90‑602. Restart NTP’s wireless studies now. This isn’t a “pause.” It’s defiance of a federal mandate. The law says: “The Secretary shall establish and carry out an electronic product radiation control program…” — 21 U.S.C.
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Lymphocyte depletion is the floor; functional competence is the ceiling. Ask your doctor, what is my ALC?
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RF Safe's angle on genetic vulnerabilities to EMFs—like that CACNA1C variant ramping up calcium channel sensitivity—highlights how DNA isn't just a passive blueprint for proteins but a dynamic conductor in the orchestra of cellular electricity. They're spot on with the
@drmichaellevin @eryney_ok Totally agree that most endogenous bioelectric cues are DC-like—but new data show gene-by-field interactions you might find intriguing: https://t.co/WIampxNynq 🔹 Single-nucleotide “bioelectric dimmer switch.” 2025 NeuroImage (Sousouria et al.,
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Small millivolt changes at the S4 voltage sensor change when ion channels open and close. In immune and excitable cells, that resets potassium, calcium, and proton flux, altering NFAT and NF‑kappaB programs and mismatching the respiratory burst. Mitochondria respond with
Cells communicate with electricity as well as chemistry. Voltage‑gated ion channels open and close when a short, positively charged protein segment (called S4) moves inside the membrane’s electric field.
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“Entropic waste” defined In this context entropic waste means persistent, non‑native temporal patterning of electromagnetic fields that reduces ion timing fidelity. It is quantified by measurable shifts in channel activation midpoints and open‑time distributions, by altered
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Ion Timing Fidelity under RF exposure, from S4 voltage sensing to mitochondrial ROS and immune dysregulation. It's how nnEMFs harm you!
Oxidative stress, inflammation, and loss of immune tolerance are etiological factors in a large fraction of chronic disease. Continuous indoor exposure to pulsed RF pushes exactly those pathways, through a mechanism that operates at the same scales as the S4 voltage sensors!
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Mistimed ions and altered membrane potential are sufficient to push immune cells into pro‑inflammatory states by well‑described mtDNA‑independent pathways. mtDNA release is a powerful amplifier and a marker of chronicity, but it is not required to start the cascade.
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Conclusion: Intensive use of mobile phones has negative impact on bladder tissue as well as the other organs. Keeping a minimum level of mobile phone use makes it easy to be kept under control of diseases in which inflammation is an etiologic factor. https://t.co/owvMHijI6u
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Breaking Research (2025) — 5G, mitochondria, and immune danger signals: how a sub‑thermal 3.5 GHz exposure shifted brain mitochondrial genes in mice https://t.co/diwXkAGBu3
A March 2025 mouse study using a smartphone‑class 5G NR signal at 3.5 GHz and sub‑thermal SAR found up‑regulation of 10 of the 13 mitochondrial DNA‑encoded OXPHOS genes in cortex after 27 hours of head‑only exposure over 6 weeks. https://t.co/Ab4Q4rcNlM
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This isn't fringe; it's a testable framework gaining momentum in 2025 literature, potentially explaining EHS or rising autoimmune trends. If validated clinically, it could reshape exposure guidelines.
VULNERABILITY: Early development, pregnancy, and childhood are periods with rapid growth, high signaling plasticity, and often in proximity to nnEMFs. Changes in Vm/calcium signaling during these windows can produce larger and lasting changes in gene programs/immune set points!
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How Long Will Humanity Continue to Ignore Bioelectric Catastrophe? We are witnessing an unprecedented global crisis, one that extends beyond mere toxicological or epidemiological paradigms and into the fundamental physics of biological coherence itself.
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Full links to the peer‑reviewed papers that back each step in the chain from S4 voltage sensing → ion‑flux timing changes → oxidative stress and immune activation → autoimmune‑like outcomes
@rfsafe FULL LINKS TO PEER‑REVIEWED SOURCES S4 voltage sensing and channel gating • Wisedchaisri G et al., Cell (2019) – Resting‑state structure and gating mechanism of a voltage‑gated sodium channel: https://t.co/t3hgmmOxWK • Catterall WA, Neuron (2010) – Ion channel voltage
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This isn't fringe; it's a testable framework gaining momentum in 2025 literature, potentially explaining EHS or rising autoimmune trends. If validated clinically, it could reshape exposure guidelines.
VULNERABILITY: Early development, pregnancy, and childhood are periods with rapid growth, high signaling plasticity, and often in proximity to nnEMFs. Changes in Vm/calcium signaling during these windows can produce larger and lasting changes in gene programs/immune set points!
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Anchored to testable biology: the central risk is loss of bioelectric timing fidelity that mis‑codes immunity and amplifies via mitochondrial dysfunction. The hazard signal in controlled animal studies already aligns with the predicted tissue susceptibility.
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Elon Musk: The “Phone” Will Die—And AI Will Replace Everything In 5-6 years, the device in your pocket won’t be a phone. It’ll be an AI edge node—no apps, no OS, just real-time, hyper-personalized content generated on demand. Music, video, even conversations: all dynamically
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Dr. Casey Means: The Metabolic Roots of Alzheimer’s & Why We’re Missing the Point The rise of early-onset dementia and Alzheimer’s—tripling since 2013—isn’t a mystery. It’s a metabolic crisis. - 30% of teens now have pre-diabetes (once unheard of). - 50% of middle-aged adults
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🧠 Scientists Say Alzheimer’s May Be an Autoimmune Disease — Not a Brain Disease A bold new theory is reshaping our understanding of Alzheimer’s disease: rather than being driven by degenerating brain cells, it may stem from the brain’s own immune system turning against itself.
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New Research Exposes a Hidden Danger in Your Home: Wi-Fi Routers May Be Supercharging Toxic Mold Household mold, usually harmless, can become 600 times more toxic when exposed to Wi-Fi signals. The electromagnetic fields (EMF) trigger mold’s survival mode, causing it to release
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The Spike Protein & Chronic Inflammation: A Looming Crisis New insights reveal how the SARS-CoV-2 spike protein—persisting in tissues from the brain to the pancreas—may drive chronic inflammation via M1 macrophage activation and endothelial damage. This isn’t just about acute
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