Will you become genetically modified? I did! Here is why I think you will too. Liz Parrish: Why She Risked Everything on an Unproven Treatment via @YouTube.

This study is the first to show that psilocybin can slow aging and extend lifespan in cells and animals, even when treatment starts later in life. Psilocybin appears safe for older adults and may offer a promising new way to promote healthy aging and treat age-related diseases.

Double the risk? Variants in the homeostatic iron regulator (HFE) gene are prevalent among individuals of European ancestry and have been linked to an increased risk of dementia.

Thank you to the Super Human Network Team. She used her own body to do what most wouldn’t even consider

Tissue regeneration relies on symmetrically dividing stem cells, which either stay as stem cells or become specialized based on their location. In zebrafish sensory organs, two different cyclinD genes control the growth of stem and progenitor cells separately—one affects stem.

Since 2009, seven people with HIV have been cured after receiving bone marrow transplants, five of which used donor cells with a mutation that blocks HIV entry. While effective, this method is too risky and complex to be widely used, so researchers are turning to gene therapy as.

New research from the Piotrowski Lab, published in Nature Communications on July 14, 2025, seeks to understand how cell division is regulated to both promote regeneration of hair cells and to also maintain a steady supply of stem cells.

Liu shows- stem cell therapy for aging is limited by poor implantation, short cell lifespan, and cancer risk. Researchers modified the FOXO3 gene in human stem cells, creating stress-resistant MSCs "Seno-resistant mesenchymal progenitor cells” (SRCs) showed resistance to aging.

RT @infinitacity: 🎤 Introducing @ParrishLiz, CEO of Core contributor to Bio-Frontiers Month Sep 1 → 30 . This is wh….

Senescent osteoblasts (OBs) contribute to bone loss and osteoporosis by disrupting bone metabolism and promoting harmful signaling. Reversing or removing these aged OBs can improve bone health, making them a promising target for future treatments.

Osteoarthritis (OA) causes joint pain and damage, mainly in older adults, and current treatments only manage symptoms without stopping disease progression. New approaches like stem cell therapy, gene therapy, and advanced drug delivery aim to repair cartilage and target the.

In the aging mouse retina, the number of rhythmic genes decreases significantly, with antigen processing genes maintaining rhythmic expression while genes involved in RNA degradation and proteasome pathways lose their daily rhythms.

A new study found that rare genetic variants in telomere and non-telomere genes impact survival in idiopathic pulmonary fibrosis (IPF) and interact with common risk genes. Patients with these variants and low polygenic risk scores had worse outcomes, suggesting genetic testing.

Aging is a major risk factor for memory loss, and rising levels of complement C3 protein may play a key role in this decline. In mice, increased C3 caused memory problems and brain damage, but blocking C3 improved symptoms, suggesting it could be a target for treating age-related.

Aging populations around the Pacific Rim are pushing universities to shift their teaching focus and prioritize research in health and aging. However, these changes come amid growing uncertainty around research funding. At a recent APRU meeting, UCLA Chancellor Julio Frenk and.

This study looked at anxiety and depression in older adults with and without frailty. The results showed no major differences in how symptoms were connected or how strong those connections were between the two groups. This means treatments for anxiety and depression can be.

This study looked at people with brain injuries and found that those who developed kidney problems (AKI) had lower levels of a protein called Klotho. People with higher Klotho levels had better chances of recovery. Klotho was also a better warning sign of kidney issues than the.

This study shows that SKL-MSCs are more effective than regular MSCs at reducing EAE symptoms and brain inflammation. This may be due to their stronger ability to protect the blood-brain barrier.

In conclusion, our study reveals a link between depression and telomere length (TL), suggesting TL could be a useful biomarker and support personalized treatment. Further research is needed to explore this relationship and improve outcomes.