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De Camilli Lab Profile
De Camilli Lab

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Laboratory of Pietro De Camilli - Department of Neuroscience at Yale University

New Haven, CT
Joined December 2018
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@PDClab
De Camilli Lab
8 months
The De Camilli lab account has moved to Bluesky:
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@PDClab
De Camilli Lab
1 year
Bridge-like lipid transfer protein 3A (BLTP3A) is associated with membranes of the late endocytic pathway and is an effector of CASM
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biorxiv.org
Recent studies have identified a family of rod-shaped proteins which includes VPS13 and ATG2 and are thought to mediate unidirectional lipid transport at intracellular membrane contacts by a bridge...
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@PDClab
De Camilli Lab
1 year
A complex of the lipid transport ER proteins TMEM24 and C2CD2 with band 4.1 at cell–cell contacts
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@PDClab
De Camilli Lab
2 years
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@PDClab
De Camilli Lab
2 years
Our new preprint reveals that the interaction of the bridge-like lipid transport protein VPS13A with the lipid scramblase XK at ER-plasma membrane contacts in K562 cells correlates with erythroid differentiation @ChaseAmos15 @YaleCellBio @YaleNeuro
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biorxiv.org
Mutations of the bridge-like lipid transport protein VPS13A and of the lipid scramblase XK result in Chorea Acanthocytosis (ChAc) and McLeod syndrome (MLS) respectively, two similar conditions...
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@PDClab
De Camilli Lab
2 years
Our review on RBG motif bridge-like #lipid transport proteins of the VPS13-ATG2 family is now out in Annual Reviews of Cellular and Development Biology @AnnualReviews @YaleCellBio
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annualreviews.org
The life of eukaryotic cells requires the transport of lipids between membranes, which are separated by the aqueous environment of the cytosol. Vesicle-mediated traffic along the secretory and...
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@PDClab
De Camilli Lab
3 years
Our collaborative study with the Cao lab at Duke-NUS reporting the synergistic pathological defects of mutations in synaptojanin 1 (Synj1) and auxilin (DNAJC6), two Parkinsonism genes, is now published.
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@PDClab
De Camilli Lab
3 years
Mutations in Parkinsonism-linked endocytic proteins synaptojanin1 and auxilin have synergistic effects on dopaminergic axonal pathology
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@PDClab
De Camilli Lab
3 years
Our paper demonstrating the segregation of synaptic vesicle proteins from ATG9 when expressed in fibroblastic cells, and their properties to be clustered in different condensates by synapsin, is now out in Nature Communications @YaleCellBio @YaleNeuro https://t.co/g1plo7OnpR
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@PDClab
De Camilli Lab
3 years
And finally on Tuesday, Michael Hanna’s @lifecerealmikey poster (P2715; B327) on lipid channeling proteins that control endocytic traffic and then his talk at the minisymposium on Membrane trafficking and Organelle Homeostasis at 430pm in room 146B.
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@PDClab
De Camilli Lab
3 years
On Tuesday, check out posters from Mick Park @Sophistarete about axonal transport and synapse formation (P2789; B403) and Daehun Park @eoqkr159 (P2807; B421) about synaptic vesicle phases,
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@PDClab
De Camilli Lab
3 years
On Monday, go hear Shujun Cai @Cai_cryoET present her work on the In situ architecture of VPS13C at the microsymposium on Organelle Structure at 11:30am in Room 201 and then go discuss at her poster (P2107; B431) right after!
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@PDClab
De Camilli Lab
3 years
On Sunday, check out the poster of Xinbo Wang @XinboWang8 (P1351; B361) about membrane remodeling properties of LRRK2.
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@PDClab
De Camilli Lab
3 years
Check out the De Camilli lab at #CellBio2022! @ASCBiology @YaleCellBio
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@PDClab
De Camilli Lab
3 years
Our review for Cold Spring Harbor Perspectives in Biology on the role of lipid transport proteins at ER-contacts sites in neurodegenerative disease is now on line.
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@PDClab
De Camilli Lab
3 years
Check out our lab's newest work from @HaniehFalahati! Proximity proteomics of synaptopodin provides insight into the molecular composition of the spine apparatus of dendritic spines @YaleNeuro @YaleCellBio
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pnas.org
The spine apparatus is a specialized compartment of the neuronal smooth endoplasmic reticulum (ER) located in a subset of dendritic spines. It cons...
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@PDClab
De Camilli Lab
3 years
Our paper describing VPS13A interaction with XK at ER-PM contact sites is now out on PNAS: https://t.co/xzMms7cKRy @AGuillenSaman @YaleCellBio @YaleNeuro @KavliAtYale
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pnas.org
Chorea-acanthocytosis (ChAc) and McLeod syndrome are diseases with shared clinical manifestations caused by mutations in VPS13A and XK, respectivel...
@PDClab
De Camilli Lab
4 years
A partnership of the lipid scramblase XK and of the lipid transfer protein VPS13A at the plasma membrane https://t.co/qradY1keOg
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@PDClab
De Camilli Lab
3 years
Membrane remodeling properties of the Parkinson’s disease protein LRRK2 https://t.co/uYfAcPJXYT
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De Camilli Lab
3 years
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